Publication
Ocular neuromyotonia
| dc.contributor.author | Soares-Dos-Reis, R | |
| dc.contributor.author | Martins, AI | |
| dc.contributor.author | Brás, A | |
| dc.contributor.author | Matos, A | |
| dc.contributor.author | Bento, C | |
| dc.contributor.author | Lemos, J | |
| dc.date.accessioned | 2018-06-20T15:20:36Z | |
| dc.date.available | 2018-06-20T15:20:36Z | |
| dc.date.issued | 2018-02-21 | |
| dc.description.abstract | Ocular neuromyotonia is a rare, albeit treatable, ocular motor disorder, characterised by recurrent brief episodes of diplopia due to tonic extraocular muscle contraction. Ephaptic transmission in a chronically damaged ocular motor nerve is the possible underlying mechanism. It usually improves with carbamazepine. A 53-year-old woman presented with a 4-month history of recurrent episodes of binocular vertical diplopia (up to 40/day), either spontaneously or after sustained downward gaze. Between episodes she had a mild left fourth nerve palsy. Sustained downward gaze consistently triggered downward left eye tonic deviation, lasting around 1 min. MR scan of the brain was normal. She improved on starting carbamazepine but developed a rash that necessitated stopping the drug. Switching to lacosamide controlled her symptoms. | pt_PT |
| dc.description.version | info:eu-repo/semantics/publishedVersion | pt_PT |
| dc.identifier.citation | Pract Neurol. 2018 Feb 21. pii: practneurol-2017-001866 | pt_PT |
| dc.identifier.doi | 10.1136/practneurol-2017-001866 | pt_PT |
| dc.identifier.uri | http://hdl.handle.net/10400.4/2148 | |
| dc.language.iso | eng | pt_PT |
| dc.peerreviewed | yes | pt_PT |
| dc.subject | Perturbações da Motilidade Ocular | pt_PT |
| dc.title | Ocular neuromyotonia | pt_PT |
| dc.type | journal article | |
| dspace.entity.type | Publication | |
| oaire.citation.startPage | practneurol-2017-001866 | pt_PT |
| rcaap.rights | openAccess | pt_PT |
| rcaap.type | article | pt_PT |